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Colleges where leftie students need to Play Victim

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As if Oberlin‘s 2013 race hoax wasn’t enough, now we have the Vassar bias hoax:

Reports of bias incidents at Vassar College that involved hateful messages left on students’ doors were actually elaborate hoaxes — and the perpetrator is none other than the student member of the Bias Incident Response Team…

This fall semester at the liberal arts college in New York saw a curiously high number of bias incident reports…The task force had one student member…who is transgendered and was also a vice president of the Vassar Student Association (VSA)…

The DC has learned that one of the perpetrators was none other than [that student].

Via Ace, who goes along with Damn Dirty Rino in suggesting that Münchausen syndrome may be at play.

I’ve made my own assumption that the student must be politically “progressive”; if it’s incorrect, let us know in the comments.

Comments

  1. What strikes me is how flat-footed awful the progressive college social justice coercion method has turned out to be. There are intervention and reeducation and indoctrination and political correctness facilitation squads behind every bush and still there is need for more.

    The Office of Political Correctness stands ready to field its crack “Bias Incident Response Team” on a moment’s notice. All hands on deck, a report has come in that the “n” word was heard emanating from the mouth of a WASP reading Huckleberry Finn aloud to ….. another WASP of a different sex.

    Okay, so maybe things are slow in the Bias Incident Response Team universe. Maybe the sheeple don’t get it. Time to call some attention to what going on in every nook and cranny, but going unreported. Hey! Look at me! Over here! I’m you Bias Incident Responder, armed and ready to take your word for whatever you claim. Give us a whistle. We are here to serve.

  3. heliotrope, thanks. That was great

    ILC. . . .

    From Wiki on hair loss and baldness

    Stem cell therapy
    Although follicles were previously thought gone in areas of complete baldness, they are more likely dormant, as recent studies have shown the scalp contains the stem cells from which the follicles arose. Research on these follicular stem cells may lead to successes in treating baldness through hair multiplication (HM), also known as hair cloning.
    One of the groups developing hair multiplication is Aderans Research Institute (ARI), a Japanese owned company in the United States. In 2008, Intercytex announced results of a Phase II trial to clone hair follicles from the back of the neck, multiply them and then reimplant the cells into the scalp. Initial testing showed at least two thirds of male patients regrew hair. The company estimated treatment would take “a number of years to complete” Phase III trials. After failing to achieve success in their trials, the company discontinued its hair multiplication project in 2010, with intention to sell off its assets and research. Aderans Research Institute Inc. (ARI) then acquired technology from Regenerative Medicine Assets Limited (formerly Intercytex Group plc) and is conducting Phase II clinical trials.
    Scientists grew the first artificial hair follicles from stem cells in 2010. Researchers in the study predicted that by 2015 people could grow new hair from their own stem cells, and have it surgically implanted at areas of hair loss. The lead investigator said preparations for clinical trials were “already in motion”. In their first human clinical trial, Replicel Life Sciences was able to regenerate 20% percent of hair on stem cell treated areas. Replicel is using dermal sheath cup cells instead of dermal papillae cells for multiplication, in distinction to Aderans. They will be conducting Phase II trials at the end of 2012. In early 2012 a research group demonstrated “functional hair regeneration from adult stem cells” in mouse animal models with the potential for “organ replacement regenerative therapies”.
    Genetics
    Curis and Procter & Gamble spent $1,000,000 on development of a topical hedgehog agonist for hair loss. The agent did not meet safety standards, and the program was stopped in 2007. In 2008 researchers at the University of Bonn announced they have found the genetic basis of two distinct forms of inherited hair loss. They found the gene P2RY5 causes a rare, inherited form of hair loss called hypotrichosis simplex. It is the first receptor in humans known to play a role in hair growth. Researchers found that disruption of the gene SOX21 in mice caused cyclical hair loss. Research has suggested SOX21 as a master regulator of hair shaft cuticle differentiation, with its disruption causing cyclical alopecia in mice models.Deletion of SOX21 dramatically affects hair lipids.